Background　and　objective:　Sigmoid　Sinus　(SS)　Wall　Reconstruction　(SSWR)　is　the　mainstream　treatment　for　pulsatile　tinnitus　(PT),　but　it　has　a　high　risk　of　recurrence.　The　damage　of　mending　material　is　the　key　cause　of　recurrence,　and　its　hemodynamic　mechanism　is　still　unclear.　The　purpose　of　this　study　was　to　investigate　the　hemodynamic　causes　of　mending　material　breakage.　Methods:　In　this　study,　six　patient-specific　geometric　models　were　reconstructed　based　on　the　data　of　the　computed　tomography　angiography　(CTA).　The　transient　fluid-structure　coupling　method　was　performed　to　clarify　the　hemodynamic　state　of　sigmoid　sinus　and　the　biomechanical　state　of　the　mending　material.　The　distribution　of　stress　and　displacement　and　the　flow　pattern　were　calculated　to　evaluate　the　hemodynamic　and　biomechanics　difference　at　the　mending　material　area.　Results:　The　area　of　blood　flow　impact　in　some　patients　(2/6)　was　consistent　with　the　damaged　location　of　the　mending　material.　The　average　stress　(6/6)　and　average　displacement　(6/6)　of　damaged　mending　material　were　higher　than　those　of　complete　mending　material.　All　(6/6)　patients　showed　that　the　high-stress　and　high-displacement　proportion　of　the　DMM　region　was　higher　than　that　of　the　CMM　region.　Moreover,　the　average　stress　fluctuation　(6/6)　and　average　displacement　(6/6)　fluctuation　degree　of　damaged　mending　material　is　larger　than　that　of　complete　mending　material.　Conclusions:　The　impact　of　blood　and　the　uneven　stress　and　displacement　fluctuation　of　the　mending　material　may　be　the　causes　of　mending　material　damage.　High　stress　and　high　displacement　might　be　the　key　causes　of　the　mending　material　damage.　©　2024　Elsevier　B.V.